一、文章
1. Tubulointerstitial nephritis antigen-like 1 is a novel matricellular protein that promotes gastric bacterial colonization and gastritis in the setting of Helicobacter pylori infection.Cell Mol Immunol. 2023 Aug;20(8):924-940.
2. Disruption of the biorhythm in gastric epithelial cell triggers inflammation in Helicobacter pylori-associated gastritis by aberrantly regulating NFIL3 via CagA activated ERK-SP1 pathway.Cell Commun Signal. 2025 Jun 15;23(1):285.
3. PD-1 does not mark tumor-infiltrating CD8+ T cell dysfunction in human gastric cancer.J Immunother Cancer. 2020, 8(2):e000422.
4. Arrestin domain containing 3 promotes Helicobacter pylori-associated gastritis by regulating protease-activated receptor 1.JCI insight, 2020, 5(15):135849.
5. Expression of ETS1 in gastric epithelial cells positively regulate inflammatory response in Helicobacter pylori-associated gastritis.Cell Death Dis, 2020, 11(7):498.
6. Cellular senescence defining the disease characteristics of Crohn's disease.Front Immunol. 2025 Jun 30:16:1616531.
7. Helicobacter pylori-Induced Angiopoietin-Like 4 Promotes Gastric Bacterial Colonization and Gastritis.Research(Wash D C). 2024 Jul 17;7:0409.
8. Tumor-infiltrating mast cells stimulate ICOS+ regulatory T cells through an IL-33 and IL-2 axis to promote gastric cancer progression.J Adv Res. 2023 Apr 20;S2090-1232(23)00119-4.
9. FasL+ PD-L2+ Identifies a Novel Immunosuppressive Neutrophil Population in Human Gastric Cancer That Promotes Disease Progression.Adv Sci(Weinh). 2022 Feb;9(5):e2103543.
10. Tumour-activated neutrophils in gastric cancer foster immune suppression and disease progression through GM-CSF-PD-L1 pathway.Gut, 2017, 66(11): 1900-1911.
二、项目
1. 主持重庆市自然科学基金（重庆市科技发展基金会）面上项目，2024NSCQ-KJFZMSX0273，幽门螺杆菌激活“CagA-CDH3-Wnt/β-catenin”轴驱动胃黏膜肠化生的作用和机制研究。
2. 主持重庆市渝中区市级引导区县科技发展基金项目（自然科学基金），20240117，细菌感染驱动炎性反馈通路上调HMOX1介导胃黏膜上皮损伤促进幽门螺杆菌感染相关慢性萎缩性胃炎进展的作用和机制研究。
3．主持国家自然科学基金青年项目，82200620，胃上皮源性生物节律分子NFIL3表达失衡在幽门螺杆菌感染致胃黏膜炎症反应中的作用及机制研究。
4. 获批重庆市人民医院百名学科人才拔尖人才项目
